Article SCOPUS
PLOS ONE  (2012)

Developmental programming of cardiovascular dysfunction by prenatal hypoxia and oxidative stress

Giussani D.A.; Camm E.J.; Niu Y.; Richter, H. G.; Blanco C.E.; Gottschalk R.; Blake E.Z.; Horder K.A.; Thakor, A. S.; Hansell, J. A.; Kane A.D.; Wooding F.B.P.; Cross C.M.; Herrera, E. A.

Keywords: model, acid, muscle, age, rat, localization, thickening, resistance, heart, distribution, animals, expression, arteries, exposure, rats, protein, protection, heat, hypoxia, pregnancy, disease, stress, mechanism, association, gestational, antioxidant, experiment, contraction, artery, cardiovascular, diseases, male, fetus, delayed, pathophysiology, outcome, tissue, wall, contractility, female, shock, drug, article, anoxia, function, activity, arterial, effects, controlled, prenatal, vascular, animal, oxidative, studies, longitudinal, study, rattus, nitrotyrosine, sympathetic, nonhuman, Ascorbic, femoral, staining, Myocardial, progeny