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Adriana Victoria Treuer Moya

Postdoctoral Researcher

Universidad de Talca

Talca, Chile

Líneas de Investigación


HETEROARYL-ACRYLONITRILE DERIVATIVES AS INHIBITORS OF NADPH OXIDASES IN AN ISOFORM-SPECIFIC MANNER. OXIDATIVE AND NITROSATIVE STRESS IN DYSTROPHIC CARDIOMYOPATHY: ROLE FOR NOX2. NOS1 RE-COUPLING AS A THERAPEUTIC STRATEGY FOR DIABETIC CARDIOMYOPATHY.

Educación

  •  Licenciatura en Biología, PONTIFICIA UNIVERSIDAD CATOLICA DE CHILE. Chile, 2003
  •  Biologa, mención Bioprocesos, Pontificia Universidad Católica de Chile. Chile, 2003
  •  Investigación y Desarrollo de Productos Bioactivos, UNIVERSIDAD DE TALCA. Chile, 2017

 

Article (23)

Maresin 1, a Proresolving Lipid Mediator, Ameliorates Liver Ischemia-Reperfusion Injury and Stimulates Hepatocyte Proliferation in Sprague-Dawley Rats
NOX Inhibition Improves beta-Adrenergic Stimulated Contractility and Intracellular Calcium Handling in the Aged Rat Heart
Synthesis of New (E)-2-(1HIndol- 3-ilcarbonil)-3-heteroaryl-acrilonitryles Via Microwave-Assisted Knoevenagel Condensation
Nitroso-Redox Crosstalk in Diabetic Cardiomyopathy
Synthesis and in silico analysis of the quantitative structure–activity relationship of heteroaril–acrilonitriles as AChE inhibitors.
Nitric oxide synthases, S-nitrosylation and cardiovascular health=> from molecular mechanisms to therapeutic opportunities (review).
Effect of Tomato Industrial Processing (Different Hybrids, Paste, and Pomace) on Inhibition of Platelet Function In Vitro, Ex Vivo, and In Vivo
NADPH oxidase-2 inhibition restores contractility and intracellular calcium handling and reduces arrhythmogenicity in dystrophic cardiomyopathy.
NOS1AP modulates intracellular Ca2+ in cardiac myocytes and is up-regulated in dystrophic cardiomyopathy
NOS1AP modulates intracellular Ca2+ in cardiac myocytes and is up-regulated in dystrophic cardiomyopathy
Activation of growth hormone releasing hormone (GHRH) receptor stimulates cardiac reverse remodeling after myocardial infarction (MI).
Dynamic denitrosylation via S-nitrosoglutathione reductase regulates cardiovascular function
Dynamic denitrosylation via S-nitrosoglutathione reductase regulates cardiovascular function.
Increased potency of cardiac stem cells compared with bone marrow mesenchymal stem cells in cardiac repair.
Effects of combination of proliferative agents and erythropoietin on left ventricular remodeling post-myocardial infarction.
Altered renal expression of angiotensin II receptors, renin receptor, and ACE-2 precede the development of renal fibrosis in aging rats.
Cardioprotective effects of growth hormone-releasing hormone agonist after myocardial infarction.
Impaired S-nitrosylation of the ryanodine receptor caused by xanthine oxidase activity contributes to calcium leak in heart failure.
Neuronal nitric oxide synthase in heart mitochondria=> a matter of life or death.
S-Nitrosylation of cardiac ion channels.
Differential role of S-nitrosylation and the NO-cGMP-PKG pathway in cardiac contractility
Deficient ryanodine receptor S-nitrosylation increases sarcoplasmic reticulum calcium leak and arrhythmogenesis in cardiomyocytes.
Reduced neuronal nitric oxide synthase expression contributes to cardiac oxidative stress and nitroso-redox imbalance in ob/ob mice.

Abstract (1)

Role of cGMP in the response to ATP in oviductal ciliated cells

Proyecto (2)

NOS1 RE-COUPLING AS A THERAPEUTIC STRATEGY FOR DIABETIC CARDIOMYOPATHY
OXIDATIVE AND NITROSATIVE STRESS IN DYSTROPHIC CARDIOMYOPATHY=> ROLE FOR NOX2
5
Adriana Treuer

Postdoctoral Researcher

BASIC BIOMEDICAL SCIENCES

Universidad de Talca

Talca, Chile

3
DANIEL GONZALEZ

PROFESOR ASOCIADO

CIENCIAS BASICAS BIOMEDICAS

UNIVERSIDAD DE TALCA

Talca, Chile

1
Jessica Zuñiga

Profesor Conferenciante

UNIVERSIDAD DE TALCA

Talca, Chile

1
Roberto Fuentealba

Docente investigador

Salud

Universidad Santo Tomás

Talca, Chile

1
IVAN CASTILLO

PATOLOGO

HOSPITAL REGIONAL DE TALCA

TALCA, Chile