Man

Pia Maria Vidal Vera

assistant professor

Universidad Católica de la Santisima Concepción

Concepcion, Chile

Líneas de Investigación


My research is focused on studying the interaction between the immune response and the central nervous system following trauma, with special emphasis in how the interaction between the two of them might play a role in the recovery of motor functions.

Educación

  •  Bachelor of Biological Sciences, UNIVERSIDAD DE CONCEPCION. Chile, 2006
  •  Bachelor of Bioengineering, UNIVERSIDAD DE CONCEPCION. Chile, 2008
  •  Biomedical Sciences, UNIVERSITEIT MAASTRICHT. Bélgica, 2013

Experiencia Académica

  •   Postdoctoral fellow Full Time

    Biomed Institute

    Hasselt, Bélgica

    2013 - 2013

  •   Postdoctoral fellow Full Time

    University Health Network

    Toronto, Canada

    2014 - 2017

  •   Postdoctoral fellow Full Time

    Fundacion Ciencia & Vida

    Santiago, Chile

    2018 - 2019

  •   assistant professor Full Time

    UNIVERSIDAD CATOLICA DE LA SANTISIMA CONCEPCION

    Concepcion, Chile

    2019 - At present

Experiencia Profesional

  •   Postdoctoral fellow Full Time

    BIOMED

    Hasselt, Bélgica

    2013 - 2013

  •   Postdoctoral fellow Full Time

    UHN

    Canada

    2014 - 2017

  •   Postdoctoral fellow Full Time

    Fundación Ciencia y Vida

    Chile

    2018 - 2019

  •   Assistant professor Full Time

    UCSC

    Chile

    2019 - At present

Premios y Distinciones

  •   Basic research paper

    Cervical Spine Research Society

    Canada, 2016

    Third place basic research paper at the 44th annual meeting

  •   Trainee professional developmental award

    Society for Neuroscience

    Estados Unidos, 2016

    Trainee professional developmental award from the Society for Neuroscience at 46th annual meeting

  •   abstract prize

    UNIVERSITY OF TORONTO

    Canada, 2016

    Second Place Abstract Prize at SpineFest 2016

  •   Basic research paper

    Cervical Spine Research Society

    Estados Unidos, 2015

    Second Place Basic research Paper at the 43th Annual meeting of the Cervical Spine Research Society

  •   Travel grant

    R&D Systems

    Estados Unidos, 2015

    R&D Systems Travel Grant to attend 45th Annual meeting of the Society for Neuroscience

  •   travel grant

    European school of Neuroimmunology

    Italia, 2015

    Travel grant award from the European School of Neuroimmunology (ESNI)

  •   travel grant

    office of research trainees

    Canada, 2015

    Travel grant award from the Office of Research Trainees (ORT) to attend the 15th ESNI Course

  •   poster prize

    Belgian society of cell and developmental biology

    Bélgica, 2013

    Poster prize Autumn meeting Belgian Society of Cell and developmental Biology

  •   travel grant

    Belgian Society of Neuroscience (BSN)

    Chile, 2012

    Travel grant award from Belgian Society of Neuroscience (BSN) for 8th FENS forum of Neuroscience meeting

  •   travel grant

    doctoral school university of Hasselt

    Chile, 2012

    Travel grant award from Doctoral School University of Hasselt for 8th FENS forum of Neuroscience meeting

  •   travel grant

    International Society of Neuroimmunology (ISNI)

    Italia, 2010

    Travel grant award from the International Society of Neuroimmunology (ISNI). 10th international congress of Neuroimmunology


 

Article (23)

Gut Microbiota-Brain Axis as a Potential Modulator of Psychological Stress after Spinal Cord Injury
Macrophage-based delivery of interleukin-13 improves functional and histopathological outcomes following spinal cord injury
Gut Microbiota Interaction with the Central Nervous System throughout Life
IgM Immunoglobulin Influences Recovery after Cervical Spinal Cord Injury by Modulating the IgG Autoantibody Response.
Mir21 modulates inflammation and sensorimotor deficits in cervical myelopathy: data from humans and animal models
The Cross-Talk Between the Dopaminergic and the Immune System Involved in Schizophrenia
ADAM17-deficiency on microglia but not on macrophages promotes phagocytosis and functional recovery after spinal cord injury
Endogenous Interleukin-10 Deficiency Exacerbates Vascular Pathology in Traumatic Cervical Spinal Cord Injury.
Targeting the Dopaminergic System in Autoimmunity.
The changes in systemic monocytes in humans undergoing surgical decompression for degenerative cervical myelopathy may influence clinical neurological recovery
Methylprednisolone treatment enhances early recovery following surgical decompression for degenerative cervical myelopathy without compromise to the systemic immune system
Delayed decompression exacerbates ischemia-reperfusion injury in cervical compressive myelopathy.
The effect of animal weight on over ground locomotion in neurological diseases with a focus in CatWalk gait assessment
Mast cells promote scar remodeling and functional recovery after spinal cord injury via mouse mast cell protease 6.
The Complement Receptor C5aR Has a Dual, Time-Dependent Effect on the Outcome of Spinal Cord Injury.
Glycine receptors control the generation of projection neurons in the developing cerebral cortex.
Immunopharmacological intervention for successful neural stem cell therapy=> New perspectives in CNS neurogenesis and repair.
Mast cells protect from post-traumatic spinal cord damage in mice by degrading inflammation-associated cytokines via mouse mast cell protease 4.
Oncostatin M reduces lesion size and promotes functional recovery and neurite outgrowth after spinal cord injury.
ADAM17 is a survival factor for microglial cells in vitro and in vivo after spinal cord injury in mice.
Glycine receptor α2 subunit activation promotes cortical interneuron migration.
Late blocking of peripheral TNF-α is ineffective after spinal cord injury in mice.
The role of "anti-inflammatory" cytokines in axon regeneration.

Proyecto (6)

Epigenetic control of transcriptional networks promoting the pathogenic behavior of CD4+ T-cells in inflammatory bowel disease
Attenuation of post-ischemia inflammation to inhibit secondary injury after ischemia-reperfusion injury following surgical decompression for degenerative cervical myelopathy
Therapeutic Approaches to Protect against Ischemia/reperfusion Injury Following Surgical Decompression for Cervical Spondylotic Myelopathy(CSM)=> A Potential Solution to Attenuate Perioperative Neurological Complications following Decompressive Surgery
Therapeutic Approaches to Protect Against Ischemia/Reperfusion Injury Following Surgical Decompression for Cervical Spondylotic Myelopathy=> A Potential Solution to Attenuate Perioperative Neurological Complication Following Decompressive Surgery
TNF-alpha converting enzyme (TACE) is a major survival factor after CNS trauma
The role of TNF-a, ADAM17 and IL-13 in CNS injury and repair

Review (1)

Targeting the Dopaminergic System in Autoimmunity
10
Pia Vidal

assistant professor

Universidad Católica de la Santisima Concepción

Concepcion, Chile

2
Rodrigo Pacheco

Associate Investigator; Head of the Laboratory of Neuroimmunology

Fundación Ciencia para la Vida

Santiago, Chile