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Daniela Paz Ponce de la Vega

Coordinadora de investigación

Universidad de Chile

Santiago, Chile

Colaboradora en investigaciones básicas y clínicas asociadas al estudio del deterioro cognitivo y el envejecimiento.

Biotecnología , UNIVERSIDAD DE CHILE. Chile, 2011

Asistente de Investigación Full Time

UNIVERSIDAD DE CHILE

Medicina

Santiago, Chile

2011 - 2014

Participación en capacitaciones para la ejecución de proyectos de investigación FONIS y FONDEF.

Participación en seminarios y conferencias de difusión de proyectos FONIS. FONDEF y FONDECYT.


Cancer Imprints an Increased PARP-1 and p53-Dependent Resistance to Oxidative Stress on Lymphocytes of Patients That Later Develop Alzheimers Disease Rogers, Nicole K.; Ponce, Daniela P. Article ISI SCOPUS FRONTIERS IN NEUROSCIENCE (2018)
Mitochondrial permeability transition pore contributes to mitochondrial dysfunction in fibroblasts of patients with sporadic Alzheimers disease Jose Perez, Maria; Quintanilla, Rodrigo A. Article ISI SCOPUS REDOX BIOLOGY (2018)
Mitochondrial Bioenergetics Is Altered in Fibroblasts from Patients with Sporadic Alzheimer's Disease Pérez, María J.; Ponce, Daniela P.; Osorio-Fuentealba, Cesar Article ISI SCOPUS Frontiers in Neuroscience (2017)
PARP-1 and p53 Regulate the Increased Susceptibility to Oxidative Death of Lymphocytes from MCI and AD Patients Rogers, NK Article ISI SCOPUS FRONTIERS IN AGING NEUROSCIENCE (2017)
Increased susceptibility to oxidative death of lymphocytes from Alzheimer patients correlates with dementia severity. Article Current Alzheimer Research (2014)
Inverse Susceptibility to Oxidative Death of Lymphocytes Obtained From Alzheimer's Patients and Skin Cancer Survivors: Increased Apoptosis in Alzheimer's and Reduced Necrosis in Cancer Behrens, M. I.; Silva, M; Salech, F; Merino D.; Sinning, M; Xiong, C. Article ISI SCOPUS Journals of Gerontology - Series A Biological Sciences and Medical Sciences (2012)
Inverse susceptibility to oxidative death of lymphocytes obtained from Alzheimer's patients and skin cancer survivors=> increased apoptosis in Alzheimer's and reduced necrosis in cancer. Behrens MI, Silva M; Salech F, Ponce DP; Merino D, Sinning M; Xiong C, Roe CM Article J Gerontol A Biol Sci Med Sci. (2012)
CK2 functionally interacts with AKT/PKB to promote the β-catenin-dependent expression of survivin and enhance cell survival. Ponce DP, Yefi R; Cabello P, Maturana JL; Niechi I, Silva E; Galindo M, Antonelli M; Marcelain K, Armisen R Article Mol Cell Biochem (2011)
Phosphorylation of AKT/PKB by CK2 is necessary for the AKT-dependent up-regulation of beta-catenin transcriptional activity. Ponce DP, Maturana JL; Cabello P, Yefi R; Niechi I, Silva E; Armisen R, Galindo M; Antonelli M, Tapia JC Article J Cell Physiol (2011)
Protein kinase CK2 promotes cancer cell viability via up-regulation of cyclooxygenase-2 expression and enhanced prostaglandin E2 production. Yefi R, Ponce DP; Niechi I, Silva E; Cabello P, Rodriguez DA; Marcelain K, Armisen R; Quest AF, Tapia JC. Article J Cell Biochem (2011)