Man

Claudio Alejandro Cabello Verrugio

Full Professor

UNIVERSIDAD ANDRES BELLO

Santiago, Chile

Líneas de Investigación


SKELETAL MUSCLE ATROPHY; AGING; FRAGILITY; RENIN ANGIOTENSIN SYSTEM; FISIOLOGIA MUSCULAR; FIBROSIS; TGF-beta FAMILY GROWTH FACTORS, SIGNALING AND REGULATION, CELL BIOLOGY.

Educación

  •  Cell Biology, P. Universidad CAtolica de Chile. Chile, 2007
  •  Biochemist, Universidad de Chile. Chile, 2001

Experiencia Académica

  •   Post doc Part Time

    PONTIFICIA UNIVERSIDAD CATOLICA DE CHILE

    Chile

    2007 - 2011

  •   Associate Investigator Full Time

    Universidad del Desarrollo

    Facultad de Medicina

    Santiago, Chile

    2011 - 2012

  •   Associate professor Full Time

    Universidad Andres Bello

    Ciencias Biologicas

    Chile

    2012 - 2018

  •   Full Professor Full Time

    Universidad Andres Bello

    Life Sciences

    Santiago, Chile

    2018 - A la fecha

Experiencia Profesional

  •   Post Doc Part Time

    Universidad Catolica de Chile

    Santiago, Chile

    2007 - 2011

  •   Associate Investigator Full Time

    Universidad del Desarrollo

    Santiago, Chile

    2011 - 2012

  •   Associate professor Full Time

    Universidad Andres Bello

    Santiago, Chile

    2012 - A la fecha

  •   Investigador Asociado

    Universidad Católica de Chile

    Santiago, Chile

    2001 - 2002

Formación de Capital Humano


Post-grado:

Co-dirección de Tesis de Doctorado Srta. Maria José Acuña, Programa de Doctorado en Ciencias Biológicas Mención Biología Celular y Molecular, P. Universidad Católica de Chile. Finalizada Enero 2013. "Papel del Sistema Renina Angiotensina en la fibrosis muscular esquelética"

Dirección de Tesis de Magister carrera de Ingeniería en Biotecnología (Facultad de Ciencias Biológicas, Universidad Andrés Bello). Sr. Franco Cisternas. Finalizada Marzo 2014.
"Efecto del eje Ang (1-7)/Mas-1 sobre la atrofia muscular esquelética: importancia de la señalización de AKT".

Dirección de Tesis de Magister carrera de Ingeniería en Biotecnología (Facultad de Ciencias Biológicas, Universidad Andrés Bello). Srta. Carla Meneses. Finalizada Abril 2014.
"Angiotensina 1-7, a través de su receptor Mas1, disminuye la apoptosis inducida por Angiotensina II en musculo esquelético"

Dirección de Tesis de Magister carrera de Ingeniería en Biotecnología (Facultad de Ciencias Biológicas, Universidad Andrés Bello). Sr. Javier Aravena Castro. Finalizada Marzo 2016.
"Regulación de la actividad biológica dependiente de miostatina por Angiotensina 1-7: implicancias en atrofia muscular esquelética"

Dirección de Tesis de Magister carrera de Bioquímica (Universidad Andrés Bello). Sr. Fabián Campos. Finalizada Marzo 2017. "Papel del receptor TGR-5 de ácidos biliares en la caquexia inducida por un modelo de enfermedad hepática crónica”.

Dirección de Tesis de Magister carrera de Ingeniería en Biotecnología (Facultad de Ciencias de la Vida, Universidad Andrés Bello). Sr. Francisco Gonzalez. En curso. "Los ácidos biliares producen atrofia muscular esquelética en forma dependiente de NF-kB”.

Dirección de Tesis de Magister carrera de Bioquímica (Facultad de Ciencias de la Vida, Universidad Andrés Bello). Sr. Luis Maldonado. En curso. "Rol de los ácidos biliares Cólico y Desoxicólico y se receptor TGR5 en la fibrosis muscular esquelética inducida por TGF-b”.

Dirección de Tesis de Doctorado Sr. Juan Carlos Rivera, Programa de Doctorado en Biociencias Moleculares, Universidad Andres Bello. Finalizada Enero 2019. "Regulación de la autofagia por Angiotensina (1-7) en un modelo de atrofia muscular esquelética inducida por lipopolisacárido"

Dirección de Tesis de Doctorado Srta. Johanna Abrigo Leon, Programa de Doctorado en Biociencias Moleculares, Universidad Andres Bello. En curso. “Efecto de los ácidos biliares a través de su receptor TGR5 sobre la atrofia muscular y función mitocondrial en el músculo esquelético en un modelo de enfermedad hepática crónica.”


Pre-grado:

Dirección de Memoria de Investigación carrera de Bioquímica (Facultad de Ciencias Químicas y Farmacéuticas, Universidad de Chile). Srta. Paula Painemal. Finalizada Mayo 2011.
"Efecto de TGF-b sobre los niveles proteicos de los receptores AT-1 y AT-2 para Angiotensina II en células musculares esqueléticas y musculo esquelético"

Dirección de Memoria de Investigación carrera de Bioquímica (Facultad de Ciencias Químicas y Farmacéuticas, Universidad Católica de Valparaíso). Srta. Johanna Ábrigo. Finalizada Mayo 2014. "TGF-?1 aumenta la expresión de atrogina-1 y MuRF-1 mediante un mecanismo dependiente de especies reactivas de oxígeno inducidas por NADPH oxidasa en células musculares esqueléticas".

Dirección de Tesis de Pregrado carrera de Kinesiología (UMCE). Sr. Ignacio Barría Olmedo. Finalizada Diciembre 2015. "Asociación de los polimorfismos de ECA-2 1075A/G, 879OA/G y 16854G/C con la generación de fuerza muscular en adultos mayores sedentarios".

Dirección de Tesis de Pregrado carrera de Kinesiología (UMCE). Sr. Alejandro Contreras Sanchez. Finalizada Diciembre 2015. "Asociación de los polimorfismos de ECA-2 1075A/G, 879OA/G y 16854G/C con la generación de fuerza muscular en adultos mayores sedentarios".

Dirección de Tesis de Pregrado carrera de Ingeniería en Biotecnología (U de Chile). Sr. Sebastian Perez. Finalizada Enero 2017. "Efecto de Angiotensina (1-7) sobre la capacidad miogénica de células satélite adultas".

Dirección de Tesis de Pregrado carrera de Tecnología Médica (Universidad Pedro de Valdivia). Sr. Betzabé Irigoyen. Finalizada Enero 2017. "Papel de eje GH/IGF-1 en la debilidad muscular esquelética inducida por hígado graso no alcohólico”.

Dirección de Unidad de Investigación carrera de Tecnología Médica (Universidad Andrés Bello). Sr. Tabita Marín. Finalizada Agosto 2016. "Estudio de Apoptosis Mionuclear en un modelo de atrofia muscular esquelética inducida por enfermedad hepática crónica”.

Dirección de Unidad de Investigación carrera de Tecnología Médica (Universidad Andrés Bello). Sr. Estefany Olguin. Finalizada Agosto 2016. "Expresión de la Desubiquitinasa 19 (Usp19) en músculo esquelético atrófico inmovilizado”.


Difusión y Transferencia


Charla Magistral Sesión Inaugural año Académico de Programas de Magíster Universidad Bernardo O´Higgins: “Más que una pasión: la importancia de hacer ciencia en Chile”. 2017

Clase Inaugural, Curso de Postgrado “Modelos Biológicos Utilizados en Biología y Fisiología Celular”. Programa de Apoyo Centro de Envejecimiento y Regeneración (CARE), Facultad de Ciencias Biológicas, P. Universidad Católica de Chile y Programa de Doctorado Facultad de Ciencias de la Salud, Universidad de Antofagasta. Enero 2011.

Clase Magistral XII Jornada Estudiantil de Kinesiología UMCE 2011.

Asistencia Diálogos con el Conocimiento: “La Genética: del debate científico al debate social”. Facultad de Medicina, Universidad de Chile. 2011.

1 Charla Explora año 2015.
3 Charlas Explora año 2017.
Participación Tour de la Ciencia (Organizado Centro Estudiantes Bioquimica UNAB).
1 Entrevistas Portales Ciencia Mexicano 2017
1 Entrevista Radial Universidad de Chile 2016.
1 Charla Instituto Chileno-Frances (Colaboración Ecos Conicyt) 2017.



 

Article (93)

Disseminated intravascular coagulation phenotype is regulated by the TRPM7 channel during sepsis
Effect of Dietary Supplements with omega-3 Fatty Acids, Ascorbic Acid, and Polyphenolic Antioxidant Flavonoid on Gene Expression, Organ Failure, and Mortality in Endotoxemia-Induced Septic Rats
NEDD4-1 deficiency impairs satellite cell function during skeletal muscle regeneration
Procoagulant phenotype induced by oxidized high-density lipoprotein associates with acute kidney injury and death
Sepsis-Induced Coagulopathy Phenotype Induced by Oxidized High-Density Lipoprotein Associated with Increased Mortality in Septic-Shock Patients
The Human Dermis as a Target of Nanoparticles for Treating Skin Conditions
Ursodeoxycholic acid induces sarcopenia associated with decreased protein synthesis and autophagic flux
Bile Acids Induce Alterations in Mitochondrial Function in Skeletal Muscle Fibers
Combined Administration of Andrographolide and Angiotensin- (1-7) Synergically Increases the Muscle Function and Strength in Aged Mice
Intensive care unit-acquired weakness: A review from molecular mechanisms to its impact in COVID-2019
Oxidized High-Density Lipoprotein Induces Endothelial Fibrosis Promoting Hyperpermeability, Hypotension, and Increased Mortality
Cholic acid and deoxycholic acid induce skeletal muscle atrophy through a mechanism dependent on TGR5 receptor
Impact of exercise training on the sarcopenia criteria in non-alcoholic fatty liver disease: a systematic review and meta-analysis
Redox-Dependent Effects in the Physiopathological Role of Bile Acids
Scaffold biomaterials and nano-based therapeutic strategies for skeletal muscle regeneration
The Critical Role of Oxidative Stress in Sarcopenic Obesity
Angiotensin (1-7) decreases myostatin-induced NF-kb signaling and skeletal muscle atrophy
Angiotensin-(1-7) Prevents Lipopolysaccharide-Induced Autophagy via the Mas Receptor in Skeletal Muscle
N-Acetyl Cysteine Attenuates the Sarcopenia and Muscle Apoptosis Induced by Chronic Liver Disease
Protective Effect of Angiotensin 1-7 on Sarcopenia Induced by Chronic Liver Disease in Mice
Role of Oxidative Stress in Hepatic and Extrahepatic Dysfunctions during Nonalcoholic Fatty Liver Disease (NAFLD)
Sarcopenia Induced by Chronic Liver Disease in Mice Requires the Expression of the Bile Acids Membrane Receptor TGR5
SARS-CoV-2/Renin-Angiotensin System: Deciphering the Clues for a Couple with Potentially Harmful Effects on Skeletal Muscle
TRPM Channels in Human Diseases
TRPM7 mediates kidney injury, endothelial hyperpermeability and mortality during endotoxemia
Circulating Endothelial Cells From Septic Shock Patients Convert to Fibroblasts Are Associated With the Resuscitation Fluid Dose and Are Biomarkers for Survival Prediction
Endothelial Cells Exhibit Two Waves of P-selectin Surface Aggregation Under Endotoxic and Oxidative Conditions
Endotoxemia-induced endothelial fibrosis inhibition improves hypotension, tachycardia, multiple organ dysfunction syndrome, cytokine response, oxidative stress, and survival
Expression Suppression and Activity Inhibition of TRPM7 Regulate Cytokine Production and Multiple Organ Dysfunction Syndrome During Endotoxemia: A New Target for Sepsis
Mitochondrial Dysfunction in Skeletal Muscle Pathologies
OxHDL controls LOX-1 expression and plasma membrane localization through a mechanism dependent on NOX/ROS/NF-kappa B pathway on endothelial cells
The p75(NTR) neurotrophin receptor is required to organize the mature neuromuscular synapse by regulating synaptic vesicle availability
Gestational Hypothyroxinemia Affects Its Offspring With a Reduced Suppressive Capacity Impairing the Outcome of the Experimental Autoimmune Encephalomyelitis
Preventive Leptin Administration Protects Against Sepsis Through Improving Hypotension, Tachycardia, Oxidative Stress Burst, Multiple Organ Dysfunction, and Increasing Survival
Role of Oxidative Stress as Key Regulator of Muscle Wasting during Cachexia
Sarcopenia in a mice model of chronic liver disease: role of the ubiquitin-proteasome system and oxidative stress
Somatotropic Axis Dysfunction in Non-Alcoholic Fatty Liver Disease: Beneficial Hepatic and Systemic Effects of Hormone Supplementation
TGF-beta requires the activation of canonical and non-canonical signalling pathways to induce skeletal muscle atrophy
Andrographolide Ameliorates Inflammation and Fibrogenesis and Attenuates Inflammasome Activation in Experimental Non-Alcoholic Steatohepatitis
Endothelial fibrosis induced by suppressed STAT3 expression mediated by signaling involving the TGF-beta 1/ALK5/Smad pathway
Endothelial-to-mesenchymal transition=> Cytokine-mediated pathways that determine endothelial fibrosis under inflammatory conditions.
Oxidative Stress in Disease and Aging: Mechanisms and Therapies 2016.
Skeletal muscle wasting: new role of nonclassical renin-angiotensin system.
Skeletal muscle wasting=> new role of nonclassical renin-angiotensin system
The complex of PAMAM-OH dendrimer with Angiotensin (1-7) prevented the disuse-induced skeletal muscle atrophy in mice
Angiotensin-(1-7) attenuates disuse skeletal muscle atrophy in mice via its receptor, Mas.
Angiotensin-(1-7) attenuates disuse skeletal muscle atrophy in mice via its receptor, Mas.
Angiotensin-(1-7) attenuates disuse skeletal muscle atrophy via the Mas receptor.
Angiotensin-(1-7) Prevents Skeletal Muscle Atrophy Induced by Transforming Growth Factor Type Beta (TGF-beta) via Mas Receptor Activation
Angiotensin-(1-7) Prevents Skeletal Muscle Atrophy Induced by Transforming Growth Factor Type Beta (TGF-β) via Mas Receptor Activation
Endothelial-to-mesenchymal transition: Cytokine-mediated pathways that determine endothelial fibrosis under inflammatory conditions.
High Fat Diet-Induced Skeletal Muscle Wasting Is Decreased by Mesenchymal Stem Cells Administration: Implications on Oxidative Stress, Ubiquitin Proteasome Pathway Activation, and Myonuclear Apoptosis
Transforming growth factor type beta (TGF- β) requires reactive oxygen species to induce skeletal muscle atrophy
Transforming growth factor type beta (TGF-?) requires reactive oxygen species to induce skeletal muscle atrophy
Transforming growth factor type beta (TGF-β) requires reactive oxygen species to induce skeletal muscle atrophy.
Angiotensin-(1-7) decreases skeletal muscle atrophy induced by angiotensin II through a Mas receptor-dependent mechanism
Apocynin inhibits the upregulation of TGF-beta(1) expression and ROS production induced by TGF-beta in skeletal muscle cells
Endotoxin-induced skeletal muscle wasting is prevented by angiotensin-(1-7) through a p38 MAPK-dependent mechanism
Expression of the Mas receptor is upregulated in skeletal muscle wasting
Suppression of transient receptor potential melastatin 4 expression promotes conversion of endothelial cells into fibroblasts via transforming growth factor/activin receptor-like kinase 5 pathway
The angiotensin-(1-7)/Mas axis reduces myonuclear apoptosis during recovery from angiotensin II-induced skeletal muscle atrophy in mice
Transforming growth factor type-beta inhibits Mas receptor expression in fibroblasts but not in myoblasts or differentiated myotubes; Relevance to fibrosis associated to muscular dystrophies
Andrographolide attenuates skeletal muscle dystrophy in mdx mice and increases efficiency of cell therapy by reducing fibrosis
Angiotensin 1-7 Decreases Skeletal Muscle Atrophy induced by Angiotensin II through Mas Receptor Dependent Mechanism
Angiotensin 1-7/Mas-1 axis attenuates the expression and signaling of TGF-1 induced by Angiotensin II in mouse skeletal muscle
Endotoxin Induces Fibrosis in Vascular Endothelial Cells through a Mechanism Dependent on Transient Receptor Protein Melastatin 7 Activity
Endotoxin-Induced Endothelial Fibrosis Is Dependent on Expression of Transforming Growth Factors beta 1 and beta 2
Endotoxin-induced vascular endothelial cell migration is dependent on TLR4/NF-kappa B pathway, NAD(P)H oxidase activation, and transient receptor potential melastatin 7 calcium channel activity
Expression of the Mas receptor is upregulated in skeletal muscle wasting.
Increases in reactive oxygen species enhance vascular endothelial cell migration through a mechanism dependent on the transient receptor potential melastatin 4 ion channel
Oxidative stress mediates the conversion of endothelial cells into myofibroblasts via a TGF-beta 1 and TGF-beta 2-dependent pathway
Restoration of muscle strength in dystrophic muscle by angiotensin-1-7 through inhibition of TGF-beta signalling
The Ang-(1-7)/Mas-1 axis attenuates the expression and signalling of TGF-beta 1 induced by AngII in mouse skeletal muscle
The angiotensin (1-7)/Mas axis reduces myonuclear apoptosis during recovery from angiotensin II-induced skeletal muscle atrophy in mice
Inhibition of the angiotensin-converting enzyme decreases skeletal muscle fibrosis in dystrophic mice by a diminution in the expression and activity of connective tissue growth factor (CTGF/CCN-2)
Lipopolysaccharide induces a fibrotic-like phenotype in endothelial cells
Reducing CTGF/CCN2 slows down mdx muscle dystrophy and improves cell therapy
Transforming growth factor type beta 1 increases the expression of angiotensin II receptor type 2 by a SMAD- and p38 MAPK-dependent mechanism in skeletal muscle
Angiotensin II receptor type 1 blockade decreases CTGF/CCN2-mediated damage and fibrosis in normal and dystrophic skeletal muscles
Angiotensin II-induced pro-fibrotic effects require p38MAPK activity and transforming growth factor beta 1 expression in skeletal muscle cells
The Internal Region Leucine-rich Repeat 6 of Decorin Interacts with Low Density Lipoprotein Receptor-related Protein-1, Modulates Transforming Growth Factor (TGF)-beta-dependent Signaling, and Inhibits TGF-beta-dependent Fibrotic Response in Skeletal Musc
Connective tissue growth factor induction by lysophosphatidic acid requires transactivation of transforming growth factor type beta receptors and the JNK pathway
CTGF/CCN-2 over-expression can directly induce features of skeletal muscle dystrophy
Fibrotic response induced by angiotensin-II requires NAD(P)H oxidase-induced reactive oxygen species (ROS) in skeletal muscle cells
TGF-beta receptors, in a Smad-independent manner, are required for terminal skeletal muscle differentiation
Skeletal muscle cells express the profibrotic cytokine connective tissue growth factor (CTGF/CCN2), which induces their dedifferentiation
A novel modulatory mechanism of transforming growth factor-ss signaling through decorin and LRP-1
Extracellular proteoglycans modify TGF-beta bio-availability attenuating its signaling during skeletal muscle differentiation
LDL receptor-related protein (LRP) is an endocytic receptor for decorin and participates in a novel regulatory mechanism of TGF beta signaling by decorin
The low density lipoprotein receptor-related protein functions as an Endocytic receptor for decorin
Heparan sulfate proteoglycans are increased during skeletal muscle regeneration: requirement of syndecan-3 for successful fiber formation
Betaglycan expression is transcriptionally up-regulated during skeletal muscle differentiation - Cloning of murine betaglycan gene promoter and its modulation by myoD, retinoic acid, and transforming growth factor-beta
Diet-Induced Nonalcoholic Fatty Liver Disease Is Associated with Sarcopenia and Decreased Serum Insulin-Like Growth Factor-1

Abstract (5)

NFkB inhibition by Andrographolide ameliorates inflammation and fibrogenesis through inflammasome substrate depletion in experimental Non-Alcoholic Steatohepatitis (NASH)
Diet-induced nonalcoholic fatty liver disease is associated with sarcopenia and decreased serum insulin growth factor-1
Lipopolysaccharide-induced skeletal muscle atrophy is prevented by Angiotensin (1-7) via Mas receptor.
Transforming growth factor type beta 1 (TGF-beta 1) induces skeletal muscle atrophy by a mechanism dependent of NADPH oxidase-induced reactive oxygen species.
Vascular endothelial fibrosis induced by endotoxin: characteristics, mechanism and therapeutic perspectives

EditorialMaterial (3)

Oxidative Stress in Disease and Aging: Mechanisms and Therapies 2018
Role of transforming growth factor family of peptides in health and diseases
Oxidative Stress in Disease and Aging: Mechanisms and Therapies

Errata (1)

Transforming growth factor type- inhibits Mas receptor expression in fibroblasts but not in myoblasts or differentiated myotubes; Relevance to fibrosis associated to muscular dystrophies (vol 42, pg 111, 2015)

Proyecto (29)

Effect of Quillaic Acid in Sarcopenia induced by COPD patients-derived factors
Deciphering muscle-liver connections in MAFLD: experimental and clinical studies
Participation of TRPC6 and TRPM7 ion channels in the adrenergic stimulation-induced coagulation
RANTES, a new myokine that participates in sarcopenia induced by bile acids during chronic liver diseases
Role of Renin angiotensin system in myogenesis and muscle regeneration: new combined therapeutic strategies based in nanomedicine for muscular dystrophies
Role of Renin angiotensin system in myogenesis and muscle regeneration=> new combined therapeutic strategies based in nanomedicine for muscular dystrophies
Mechanisms involved in cachexia induced by chronic liver diseases
MECHANISMS INVOLVED IN THE CACHEXIA INDUCED BY CHRONIC LIVER DISEASES
Participation of TRPM7 in the generation of endothelial dysfunction characteristics, enhanced inflammatory response, and multiple organ failure during endotoxemia
PARTICIPATION OF TRPM7 IN THE GENERATION OF ENDOTHELIAL DYSFUNCTION CHARACTERISTICS, ENHANCED INFLAMMATORY RESPONSE, AND MULTIPLE ORGAN FAILURE DURING ENDOTOXEMIA.
The vascular neural muscle unit in sepsis induced intensive care unit acquired-weakness
The vascular neural muscle unit in sepsis induced intensive care unit acquired-weakness.
"Effects of Angiotensin 1-7 in Cell Death by Apoptosis on Angiotensin II-induced Skeletal Muscle Atrophy".
Angiotensina 1-7 modula la señalización dependiente del factor de crecimiento tipo insulínico 1 y el factor nuclear kappa B en un modelo de atrofia muscular esquelética inducido por lipopolisacárido
Anti-atrophic role of Angiotensin 1-7 on skeletal muscle
Effects of Angiotensin 1-7 in Cell Death by Apoptosis on Angiotensin II-induced Skeletal Muscle Atrophy
Role of Angiotensin 1-7 on skeletal muscle wasting
ANGIOTENSINA 1-7 MODULA LA SEÑALIZACION DEPENDIENTE DEL FACTOR DE CRECIMIENTO TIPO INSULINICO 1 Y EL FACTOR NUCLEAR KAPPA B EN UN MODELO DE ATROFIA MUSCULAR ESQUELETICA INDUCIDO POR LIPOPOLISACARIDO
Anti-atrophic role of Angiotensin 1-7 on skeletal muscle
Anti-atrophic role of Angiotensin 1-7 on skeletal muscle
Sepsis-induced endothelial fibrosis: Role of TRPM4 and TRPM7 ion channels as novel targets for prevention of endothelial fibrosis during sepsis
SEPSIS-INDUCED ENDOTHELIAL FIBROSIS=> ROLE OF TRPM4 AND TRPM7 ION CHANNELS AS NOVEL TARGETS FOR PREVENTION OF ENDOTHELIAL FIBROSIS DURING SEPSIS
“Anti-atrophic role of Angiotensin 1-7 on skeletal muscle”.
Novel antifibrotic role of the ACE2/Angiotensin 1-7/Mas axis in skeletal muscular dystrophies
NOVEL ANTIFIBROTIC ROLE OF THE ACE2/ANGIOTENSIN 1-7/MAS AXIS IN SKELETAL MUSCULAR DYSTROPHIES
CTGF=> the factor involved in fibrosis development in DMD
Droga Botánica para el tratamiento de enfermedades crónicas asociadas a fibrosis de alta incidencia nacional y mundial
Profibrotic effect of the renin-angiotensin system and connective tissue growth factor (CTGF) in dystrophic skeletal muscle
Mecanismos Reguladores de la Actividad Biológica de TGF-beta durante la diferenciación de células musculares esqueléticas

Review (10)

Endothelial dysfunction in pregnancy metabolic disorders
The Emerging Roles of Nicotinamide Adenine Dinucleotide Phosphate Oxidase 2 in Skeletal Muscle Redox Signaling and Metabolism
Central role of transforming growth factor type beta 1 in skeletal muscle dysfunctions: An update on therapeutic strategies
Combined Therapies for Duchenne Muscular Dystrophy to Optimize Treatment Efficacy
TGF-? and hepatocellular carcinoma: When a friend becomes an enemy
Transforming growth factor-beta family: Advances in vascular function and signaling
Renin-Angiotensin System: An Old Player with Novel Functions in Skeletal Muscle
Oxidative stress-modulated TRPM ion channels in cell dysfunction and pathological conditions in humans
Angiotensin II: Role in skeletal muscle atrophy
Novel regulatory mechanisms for the proteoglycans decorin and biglycan during muscle formation and muscular dystrophy
118
Claudio Cabello

Full Professor

Ciencias Biologicas

UNIVERSIDAD ANDRES BELLO

Santiago, Chile

53
Felipe Simon

Profesor Titular

Department of Biological Sciences

UNIVERSIDAD ANDRES BELLO FAC DE CS BIOLOGICAS, DPTO DE CS BIOLOGICAS

Santiago, Chile

32
Enrique Brandan

Profesor Titular

Biología Celular y Molecular

PONTIFICIA UNIVERSIDAD CATOLICA DE CHILE

Santiago, Chile

17
Daniel Cabrera

Investigador Adjunto

Gastroenterología

Pontificia Universidad Catolica de Chile

santiago, Chile

12
Johanna Ábrigo

Bioquímico

Centro de Bioinformática y Biología Integrativa

Santiago, Chile

11
César Echeverría

DIRECTOR DE INVESTIGACIÓN

Medicina

Universidad de Atacama

COPIAPO, Chile

6
Diego Varela

Profesor Asociado

Fisiología y Biofísica

FACULTAD DE MEDICINA, UNIVERSIDAD DE CHILE

Santiago, Chile

6
Andrea González

Docente

Pontificia Universidad Católica de Valparaíso

Valparaiso, Chile

6
María Acuña

Directora de Investigación, académico e investigador

Universidad Bernardo O´Higgins

Santiago, Chile

6
Sebastián Gatica

Investigador Postdoctoral

Instituto Milenio de Inmunología e Inmunoterapia

Santiago, Chile

5
Claudia Riedel

Full Professor

Life Sciences

Universidad Andres Bello

Santiago, Chile

5
Alvaro Elorza

FULL PROFESSOR

INSTITUTE OF BIOMEDICAL SCIENCES

UNIVERSIDAD ANDRES BELLO

Santiago, Chile

4
Cristian Vilos

Profesor Jornada Completa

Medicina

Universidad de Talca

Talca, Chile

4
MARCO ARRESE

FULL PROFESSOR

GASTROENTEROLOGY

PONTIFICIA UNIVERSIDAD CATÓLICA DE CHILE

Santiago, Chile

4
Yaneisi Vazquez

Directora técnica de proyectos

Genética Molecular

Pontificia Universidad Católica de Chile

Santiago, Chile

4
Juan Rivera

Investigador

Instituto Milenio de Inmunología e Inmunoterapia

Santiago, Chile

4
Maria Vial

Associate Professor/Profesor Asociado

Instituto de Ciencias e Innovación de Medicina

UNIVERSIDAD DEL DESARROLLO

Santiago, Chile

3
Carlos Vio

Profesor

Fisiologia

PONTIFICIA UNIVERSIDAD CATÓLICA DE CHILE

Santiago, Chile

3
Alvaro Becerra

Profesor Asociado

Ciencias Químico y Biológicas

Universidad Bernardo O'Higgins

Santiago, Chile

3
Hugo Olguin

Profesor Asociado

Biología Celular y Molecular

Pontificia Universidad Católica de Chile

Santiago, Chile

3
Pablo Tapia

Jefe Técnico Unidad de Paciente Crítico (UPC)

Unidad Paciente Critico (UPC)

Hospital Clinico Metropolitano La Florida

Santiago, Chile

3
Ricardo Armisen

Associate Professor

Centro de Genética y Genómica

Universidad del Desarrollo

Santiago, Chile

3
Hugo Olguin

Profesor Asociado

Biología Celular y Molecular

PONTIFICIA UNIVERSIDAD CATÓLICA DE CHILE

Santiago, Chile

2
Margarita Pizarro

PONTIFICIA UNIVERSIDAD CATÓLICA DE CHILE

Santiago, Chile

2
Oscar Cerda

Associate Professor

Molecular and Cellular Biology

Universidad de Chile

Santiago, Chile

2
Maria Otero

Profesor Asociado

UNIVERSIDAD ANDRES BELLO

Santiago, Chile

2
Jaime Gutierrez

Associate Professor and Researcher

Tecnología Médica

Universidad San Sebastián

Santiago, Chile

2
Juan Santibáñez

Professor of Research

Molecular Oncology

Institute for Medical Research

Belgrade, Serbia

2
Ricardo Fernández

Associate Professor

Salud

Universidad de Los Lagos

Osorno, Chile

2
lisbell estrada

Decana Facultad Ciencias de la Salud

Facultad de Ciencias de la Salud

Universidad Bernardo O Higgins

santiago, Chile

2
ricardo fadic

Jefe de Division

Division de Neurociencias

DEPARTAMENTO DE NEUROLOGÍA, FACULTAD DE MEDICINA, P. UNIVERSIDAD CATÓLICA DE CHILE

Santiago, Chile

2
Ricardo Fernandez

Profesor Asociado A

Salud

Universidad de Los Lagos

Osorno, Chile

1
Fernando Ezquer

Full Professor

Center for Regenerative Medicine

Universidad del Desarrollo

Santiago, Chile

1
Carlos Cespedes

Tecnologo Medico

Fisiologia

P. Universidad Católica de Chile

Santiago, Chile

1
Nibaldo Inestrosa

Full Professor

PONTIFICIA UNIVERSIDAD CATÓLICA DE CHILE

Santiago, Chile

1
Nicolas Pacheco

PROFESOR ADJUNTO

CIENCIAS BIOLOGICAS

Universidad Andrés Bello

Santiago centro, Chile

1
Sergio Mezzano

Catedratico

Medicina

Universidad Austral de Chile

Valdivia, Chile

1
Fernando Gonzalez

Titular Professor

Facultad de Ciencias de la Vida

Universidad Andres Bello

Santiago, Chile

1
Marcelo Andia

Associate Professor

Radiology

Pontificia Universidad Catolica de Chile

Santiago, Chile

1
Claudio Perez

Profesor Asistente

Departamento de Ciencias Fisiológicas

Pontificia Universidad Catolica de Chile

Santiago, Chile

1
Felipe Court

Full Professor and Director

Center for Integrative Biology

Universidad Mayor

Huechuraba, Chile

1
Monica Caceres

Profesor asociado

Instituto de ciencias biomedicas

Universidad de Chile

Santiago, Chile

1
Marco Fuenzalida

Full Professor

Physiology

UNIVERSIDAD DE VALPARAÍSO

Valparaíso, Chile

1
Francisca Bronfman

Full Professor

Instituto de Ciencias Biomédicas

Universidad Andrés Bello

Santiago, Chile

1
Luis Velasquez

Director and Full Professor

Medicine

Universidad SEK

Santiago, Chile

1
Maria Opazo

Profesor Asociado

Universidad de Las Américas

Santiago, Chile

1
Enrique Jaimovich

Full Professor

ICBM

FACULTAD DE MEDICINA, UNIVERSIDAD DE CHILE

Santiago, Chile

1
Claudio Hetz

Deputy Director

INSTITUTO DE CIENCIAS BIOMÉDICAS, FACULTAD DE MEDICINA, UNIVERSIDAD DE CHILE

SANTIAGO, Chile

1
VALERIA MARQUEZ

Profesor Asistente

Centro de Nanotecnologia Aplicada

Universidad Mayor de Chile

SANTIAGO, Chile

1
Xavier Figueroa

Associate Professor

Departamento de Fisiologia

PONTIFICIA UNIVERSIDAD CATÓLICA DE CHILE

Santiago, Chile

1
Maria Paz Marzolo

Profesor Titular

Departamento de Biología Celular y Molecular

PONTIFICIA UNIVERSIDAD CATOLICA DE CHILE, FAC. CIENCIAS BIOLOGICAS, DEPTO. BIOLOGIA

Santiago, Chile

1
Alexis Kalergis

Full Professor

PONTIFICIA UNIVERSIDAD CATÓLICA DE CHILE. FACULTAD DE CIENCIAS BIOLÓGICAS

Santiago, Chile

1
Arnoldo Riquelme

Profesor titular

Gastroenterología

PONTIFICIA UNIVERSIDAD CATÓLICA DE CHILE, DEPARTAMENTO DE GASTROENTEROLOGÍA

Santiago, Chile

1
Alejandro Vallejos

Asistente de Investigación

Ciencias de la Vida

Laboratorio de Fisiopatología Integrativa

Santiago, Chile

1
Mario Chiong

Profesor

Departamento de Bioquímica y Biología Molecular

Universidad de Chile

Santiago, Chile

1
JUAN PABLO HENRIQUEZ

Associate Professor

Cell Biology

UNIVERSIDAD DE CONCEPCIÓN, FACULTAD DE CIENCIAS BIOLÓGICAS, DEPARTAMENTO DE BIOLOGÍA CELULAR

Concepción, Chile

1
Pablo Cañón

Académico Regular

Escuela de Agronomía

Universidad Mayor de Chile

Santiago, Chile

1
Diego Venegas

Full Professor

Quimica de los Materiales

UNIVERSIDAD DE SANTIAGO DE CHILE, FACULTAD DE QUÍMICA Y BIOLOGÍA, DEPARTAMENTO DE QUÍMICA DE LOS MATERIALES

Santiago, Chile

1
Francisco Barrera

Profesor Asociado

Gastroenterología

Pontificia Universidad Católica de Chile

Santiago, Chile

1
Rebeca Aldunate

Profesor Titular

Ciencias

UNIVERSIDAD SANTO TOMÁS

Santiago, Chile

1
Viviana Pérez

Profesor Asistente

Fisiología

Universidad de Concepción

Concepcion, Chile

1
MIGUEL PEREZ

Associate Professor

Pontificia Universidad Catolica de Valparaiso

VALPARAISO, Chile

1
Nelson Osses

Associated Professor

Institute of Chemistry

PONTIFICIA UNIVERSIDAD CATÓLICA DE VALPARAÍSO

Valparaíso, Chile